The combination therapy with amlodipine and Atorlip, but not each monotherapy, suppresses AngII-induced AAA formation in mice in vivo, for which Rho-kinase inhibition may be involved. As articulated by the two case studies, the presence of metabolite kinetic data (i.e., norverapamil) provided unequivocal evidence in order to tease out the actual pathway responsible for the interaction between Atorlip and verapamil.
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